Associate Professor, Neurology, UCSF School of Medicine
Neurology
Dr. Adam L. Boxer, MD
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Doctor Adam L. Boxer, MD works in the field of Neurology. He attended NYU School of Medicine. Adam Boxer has written publications. He has 71 publications published with PubMed. The lastest publication is: 'Cognition and neuropsychiatry in behavioral variant frontotemporal dementia by disease stage.' Adam Boxer is registered with Medicare and accepts Medicare payments.
Publications
- Bayesian segmentation of brainstem structures in MRI.
- Dysfunctionally phosphorylated type 1 insulin receptor substrate in neural-derived blood exosomes of preclinical Alzheimer's Disease.
- A Multiancestral Genome-Wide Exome Array Study of Alzheimer Disease, Frontotemporal Dementia, and Progressive Supranuclear Palsy.
- Oxytocin for frontotemporal Dementia: A randomized dose-finding study of safety and tolerability.
- Rarity of the Alzheimer Disease-Protective APP A673T Variant in the United States.
- Effects of Multiple Genetic Loci on Age at Onset in Late-Onset Alzheimer Disease: A Genome-Wide Association Study.
- Treatment of frontotemporal Dementia.
- Altered network connectivity in frontotemporal Dementia with C9orf72 hexanucleotide repeat expansion.
- At the interface of sensory and motor dysfunctions and Alzheimer's Disease.
- Davunetide in patients with Progressive supranuclear palsy: a randomised, double-blind, placebo-controlled phase 2/3 trial.
- Divergent CSF Ä alterations in two common tauopathies: Alzheimer's Disease and progressive supranuclear palsy.
- Clinical trials: past, current, and future for atypical parkinsonian Syndromes.
- Erratum to: Modeling key pathological features of frontotemporal Dementia with C9ORF72 repeat expansion in iPSC-derived human neurons.
- The functional oculomotor network and saccadic cognitive control in healthy elders.
- An epigenetic signature in peripheral blood associated with the haplotype on 17q21.31, a risk factor for neurodegenerative tauopathy.
- Practical utility of amyloid and FDG-PET in an academic Dementia center.
- CSF neurofilament concentration reflects Disease severity in frontotemporal degeneration.
- NIH EXAMINER: Conceptualization and Development of an Executive Function Battery.
- Antisaccade task reflects cortical involvement in mild cognitive impairment.
- Demographic and Neuropsychiatric Factors Associated With Off-label Medication Use in Frontotemporal Dementia and Alzheimer's Disease.
- Modeling key pathological features of frontotemporal Dementia with C9ORF72 repeat expansion in iPSC-derived human neurons.
- Intrinsic connectivity network disruption in Progressive supranuclear palsy.
- Dissociable executive functions in behavioral variant frontotemporal and Alzheimer Dementias.
- PET Imaging of Neuropathology in Tauopathies: Progressive Supranuclear Palsy.
- TDP-43 frontotemporal lobar degeneration and autoimmune Disease.
- Neurodegenerative Disease Phenotypes in Carriers of MAPT p.A152T, A Risk Factor for Frontotemporal Dementia Spectrum Disorders and Alzheimer Disease.
- Memantine in patients with frontotemporal lobar Degeneration: a multicentre, randomised, double-blind, placebo-controlled trial.
- Criteria for the diagnosis of Corticobasal degeneration.
- Patterns of striatal Degeneration in frontotemporal dementia.
- Treatment implications of C9ORF72.
- The advantages of frontotemporal Degeneration drug development (part 2 of frontotemporal Degeneration: The next therapeutic frontier).
- Frontotemporal Degeneration, the next therapeutic frontier: Molecules and animal models for frontotemporal Degeneration drug development.
- Frontotemporal Dementia due to C9ORF72 mutations: Clinical and imaging features.
- Evidence for a role of the rare p.A152T variant in MAPT in increasing the risk for FTD-spectrum and Alzheimer's Diseases.
- Saccade abnormalities in autopsy-confirmed frontotemporal lobar degeneration and Alzheimer Disease.
- Expanded GGGGCC Hexanucleotide Repeat in Noncoding Region of C9ORF72 Causes Chromosome 9p-Linked FTD and ALS.
- The chromosome 9 ALS and FTD locus is probably derived from a single founder.
- Behavioral variant frontotemporal dementia with corticobasal degeneration pathology: phenotypic comparison to bvFTD with Pick's Disease.
- Clinicopathological correlations in Corticobasal degeneration.
- Anti-saccade performance predicts executive function and brain structure in normal elders.
- Mutual gaze in Alzheimer's Disease, frontotemporal and semantic dementia couples.
- An open-label study of memantine treatment in 3 subtypes of frontotemporal lobar Degeneration.
- Apolipoprotein E epsilon4 is associated with Disease-specific effects on brain atrophy in Alzheimer's Disease and frontotemporal dementia.
- Frontotemporal Dementia treatment: current symptomatic therapies and implications of recent genetic, biochemical, and neuroimaging studies.
- Deformation-based morphometry reveals brain atrophy in frontotemporal Dementia.
- Medial versus lateral frontal lobe contributions to voluntary saccade control as revealed by the study of patients with frontal lobe Degeneration.
- Patterns of brain atrophy that differentiate Corticobasal degeneration syndrome from progressive supranuclear palsy.
- Clinical features of frontotemporal Dementia.
- Cinguloparietal atrophy distinguishes Alzheimer Disease from semantic dementia.
- Predicting amyloid status in Corticobasal syndrome using modified clinical criteria, magnetic resonance imaging and fluorodeoxyglucose positron emission tomography.
- Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's Disease.
- Amyloid imaging in distinguishing atypical prion Disease from Alzheimer Disease.
- Executive dysfunction in hyperhomocystinemia responds to homocysteine-lowering treatment.
- Classification and prediction of clinical Alzheimer's diagnosis based on plasma signaling proteins.
- Comparison of family histories in FTLD subtypes and related tauopathies.
- Abhorring the vacuum: use of Alzheimer’s Disease medications in frontotemporal dementia.
- Focal right inferotemporal atrophy in AD with disproportionate visual constructive impairment.
- An algorithm for genetic testing of frontotemporal lobar Degeneration.
- Role of the tau gene region chromosome inversion in progressive supranuclear palsy, Corticobasal degeneration, and related disorders.
- Clinical-neuroimaging characteristics of dysexecutive mild cognitive impairment.
- Clinical and neuropsychological features of Corticobasal degeneration.
- Oculomotor function in frontotemporal lobar degeneration, related disorders and Alzheimer''s Disease.
- Clinical, neuroimaging and neuropathological features of a new chromosome 9p-linked FTD-ALS family.
- Bapineuzumab.
- Gray matter correlates of set-shifting among neurodegenerative Disease, mild cognitive impairment, and healthy older adults.
- Off-label medication use in frontotemporal Dementia.
- Visual search patterns in semantic Dementia show paradoxical facilitation of binding processes.
- Baseline predictors of clinical progression among patients with dysexecutive mild cognitive impairment.
- Neurodegenerative Disease in 2015: Targeting tauopathies for therapeutic translation.
- Cognition and neuropsychiatry in behavioral variant frontotemporal dementia by Disease stage.
Schools
New York University School Of Medicine
Procedures Preformed
- EEG (Electroencephalogram)
Conditions Treated
- Alzheimer's Disease
- Anterior Horn Disease
- Ataxia
- Cerebral Artery Thrombosis
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Doctors Specialties
- Neurology
Accepted Insurances
Education
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Stanford University -
California Pacific Medical Center -
NYU School of Medicine
Hospital
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UCSF Medical Center
Drug Facts
| NPI NUMBER | 1841231800 | |
| NPPES Provider LastName | BOXER | |
| NPPES Provider FirstName | ADAM | |
| NPPES Provider ZIPCode | 941432202 | |
| NPPES Provider State | CA | |
| Specialty Description | Neurology | |
| Total Claim Count | 133.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1841231800 |
| Last Name Of The Provider | BOXER |
| First Name Of The Provider | ADAM |
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