Professor of Medicine, Vanderbilt University
Endocrinology
Dr. Macrae Linton, MD
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Macrae Linton practices Endocrinology. He went to University of Tennessee College of Medicine. He received awards:"America's Top Doctors", "Nashville Lifestyles Magazine" and "Elected Member". Macrae Linton is also published. He has 101 publications published. The lastest publication: "On the relationship between cholesterol lowering and coronary disease event rate.' Macrae Linton, MD accepts Medicare payments and is listed with Medicare.gov.
Publications
- HDL: Beyond Atheroprotection.
- Macrophage IκB Kinase α Deficiency Suppresses Akt Phosphorylation, Reduces Cell Survival, and Decreases Early Atherosclerosis.
- Macrophage apoAI protects against dyslipidemia-induced dermatitis and atherosclerosis without affecting HDL.
- Isolevuglandin-Type Lipid Aldehydes Induce the Inflammatory Response of Macrophages by Modifying Phosphatidylethanolamines and Activating the Receptor for Advanced Gly...
- Macrophage deficiency of Akt2 reduces atherosclerosis in Ldlr null mice.
- Expression of ApoESendai From Macrophages Suppresses Atherosclerosis While Causing Lipoprotein Glomerulopathy in Hyperlipidemic Mice.
- Association of soy food intake with risk and biomarkers of coronary heart disease in Chinese men.
- Serum proprotein convertase subtilisin/kexin type 9 and cell surface low-density lipoprotein receptor: evidence for a reciprocal regulation.
- Free fatty acids are associated with metabolic syndrome and insulin resistance but not inflammation in systemic lupus erythematosus.
- Oral retinoids and plasma lipids.
- Nursing assistance for families of patients.
- Correlated motion and oscillation of neighboring cells in vitro.
- Acyl-coenzyme A:cholesterol acyltransferase promotes oxidized LDL/oxysterol-induced apoptosis in macrophages.
- Myeloid lineage cell-restricted insulin resistance protects apolipoproteinE-deficient mice against atherosclerosis.
- Failure of ACAT inhibition to retard atherosclerosis.
- Genetic abnormalities in apolipoprotein B.
- Murine bone marrow transplantation as a novel approach to studying the role of macrophages in lipoprotein metabolism and atherogenesis.
- Reduced atherosclerotic lesions in mice deficient for total or macrophage-specific expression of scavenger receptor-A.
- Macrophage lipoprotein lipase promotes foam cell formation and atherosclerosis in low density lipoprotein receptor-deficient mice.
- Massive xanthomatosis and altered composition of atherosclerotic lesions in hyperlipidemic mice lacking acyl CoA:cholesterol acyltransferase 1.
- Current perspectives on statins.
- Increased cerebral cortical lipid peroxidation and abnormal phospholipids in aged homozygous apoE-deficient C57BL/6J mice.
- Determination of the lower threshold of apolipoprotein E resulting in remnant lipoprotein clearance.
- A direct role for the macrophage low density lipoprotein receptor in atherosclerotic lesion formation.
- Macrophage lipoprotein lipase promotes foam cell formation and atherosclerosis in vivo.
- Disruption of the 12/15-lipoxygenase gene diminishes atherosclerosis in apo E-deficient mice.
- Retroviral gene therapy in ApoE-deficient mice: ApoE expression in the artery wall reduces early foam cell lesion formation.
- Macrophages, lipoprotein metabolism, and atherosclerosis: insights from murine bone marrow transplantation studies.
- Recycling of apolipoprotein E in mouse liver.
- On the relationship between cholesterol lowering and coronary disease event rate.
- Hepatic apo E expression is required for remnant lipoprotein clearance in the absence of the low density lipoprotein receptor.
- Long runs of adenines and human mutations.
- Secretion from cell culture of HDL and VLDL bearing apoB-33 with a large internal deletion.
- Leukocyte low density lipoprotein receptor (LDL-R) does not contribute to LDL clearance in vivo: bone marrow transplantation studies in the mouse.
- Reading-frame restoration by transcriptional slippage at long stretches of adenine residues in mammalian cells.
- Increased atherosclerosis in mice reconstituted with apolipoprotein E null macrophages.
- Identification and characterization of truncated forms of apolipoprotein B in hypobetalipoproteinemia.
- Familial hypobetalipoproteinemia.
- Four new mutations in the apolipoprotein B gene causing hypobetalipoproteinemia, including two different frameshift mutations that yield truncated apolipoprotein B pro...
- Transgenic mice expressing high plasma concentrations of human apolipoprotein B100 and lipoprotein(a).
- Transgenic mice expressing human apoB100 and apoB48.
- Expression of human apolipoprotein B90 in transgenic mice. Demonstration that apolipoprotein B90 lacks the structural requirements to form lipoprotein.
- Prevention of atherosclerosis in apolipoprotein E-deficient mice by bone marrow transplantation.
- Transgenic mice expressing high levels of human apolipoprotein B develop severe atherosclerotic lesions in response to a high-fat diet.
- Isoform-specific effects of apolipoprotein E on atherogenesis: gene transduction studies in mice.
- Increased cholesterol efflux in apolipoprotein AI (ApoAI)-producing macrophages as a mechanism for reduced atherosclerosis in ApoAI((-/-)) mice.
- Class A scavenger receptors, macrophages, and atherosclerosis.
- Retrovirus-mediated expression of apolipoprotein A-I in the macrophage protects against atherosclerosis in vivo.
- The inflamed plaque: cytokine production and cellular cholesterol balance in the vessel wall.
- Lack of macrophage fatty-acid-binding protein aP2 protects mice deficient in apolipoprotein E against atherosclerosis.
- Peroxisome proliferator-activated receptor-gamma agonist troglitazone protects against nondiabetic glomerulosclerosis in rats.
- A recycling pathway for resecretion of internalized apolipoprotein E in liver cells.
- Mouse models of hyperlipidemia and atherosclerosis.
- Increased atherosclerosis in LDL receptor-null mice lacking ACAT1 in macrophages.
- The cell biology and physiologic relevance of ApoE recycling.
- Expression of P1 DNA in mammalian cells and transgenic mice.
- Phenotypes of apolipoprotein B and apolipoprotein E after liver transplantation.
- Expression of carboxyl-terminally truncated forms of human apolipoprotein B in rat hepatoma cells. Evidence that the length of apolipoprotein B has a major effect on t...
- Apolipoprotein B gene mutations affecting cholesterol levels.
- Reading-frame restoration with an apolipoprotein B gene frameshift mutation.
- Cyclooxygenase-2 and atherosclerosis.
- Retroviral mediated expression of the human myeloid nuclear antigen in a null cell line upregulates Dlk1 expression.
- Cyclooxygenase-2 promotes early atherosclerotic lesion formation in LDL receptor-deficient mice.
- Cytoplasmic fatty acid-binding proteins: emerging roles in metabolism and atherosclerosis.
- Persistence of high density lipoprotein particles in obese mice lacking apolipoprotein A-I.
- Interplay between apolipoprotein E and scavenger receptor class B type I controls coronary atherosclerosis and lifespan in the mouse.
- Inhibition of cyclooxygenase with indomethacin phenethylamide reduces atherosclerosis in apoE-null mice.
- Conditional knockout of macrophage PPARgamma increases atherosclerosis in C57BL/6 and low-density lipoprotein receptor-deficient mice.
- Reduced ABCA1-mediated cholesterol efflux and accelerated atherosclerosis in apolipoprotein E-deficient mice lacking macrophage-derived ACAT1.
- The recycling of apolipoprotein E in macrophages: influence of HDL and apolipoprotein A-I.
- ACAT inhibition: bad for macrophages, good for smooth muscle cells?
- ApoE-mediated cholesterol efflux from macrophages: separation of autocrine and paracrine effects.
- ACAT1 deficiency disrupts cholesterol efflux and alters cellular morphology in macrophages.
- Reduced macrophage apoptosis is associated with accelerated atherosclerosis in low-density lipoprotein receptor-null mice.
- Quantitative and qualitative differences in proatherogenic NKT cells in apolipoprotein E-deficient mice.
- Angiotensin II amplifies macrophage-driven atherosclerosis.
- Combined adipocyte-macrophage fatty acid-binding protein deficiency improves metabolism, atherosclerosis, and survival in apolipoprotein E-deficient mice.
- Unique pathway for cholesterol uptake in fat cells.
- Proatherogenic role for NK cells revealed.
- The recycling of apolipoprotein E and its amino-terminal 22 kDa fragment: evidence for multiple redundant pathways.
- Cyclooxygenase-2 and inflammation in atherosclerosis.
- The role of fibrates in managing hyperlipidemia: mechanisms of action and clinical efficacy.
- Apolipoprotein AI as therapy for atherosclerosis: does the future of preventive cardiology include weekly injections of the HDL protein?
- Macrophages, inflammation, and atherosclerosis.
- Premature coronary-artery atherosclerosis in systemic lupus erythematosus.
- Inactivation of macrophage scavenger receptor class B type I promotes atherosclerotic lesion development in apolipoprotein E-deficient mice.
- Macrophage apolipoprotein A-I expression protects against atherosclerosis in ApoE-deficient mice and up-regulates ABC transporters.
- A practical approach to risk assessment to prevent coronary artery disease and its complications.
- Hepatocyte-derived ApoE is more effective than non-hepatocyte-derived ApoE in remnant lipoprotein clearance.
- The recycling of apolipoprotein E in primary cultures of mouse hepatocytes. Evidence for a physiologic connection to high density lipoprotein metabolism.
- Absence of angiotensin II type 1 receptor in bone marrow-derived cells is detrimental in the evolution of renal fibrosis.
- Rapid quantification of murine ABC mRNAs by real time reverse transcriptase-polymerase chain reaction.
- B-lymphocyte deficiency increases atherosclerosis in LDL receptor-null mice.
- Adipocyte fatty acid-binding protein, aP2, alters late atherosclerotic lesion formation in severe hypercholesterolemia.
- Correlates of high serum C-reactive protein levels in a socioeconomically disadvantaged population.
- Evidence of metabolic syndrome in lean children with premature pubarche at diagnosis.
- Lentiviral transduction of apoAI into hematopoietic progenitor cells and macrophages: applications to cell therapy of atherosclerosis.
- Lipoprotein subclasses and particle size determined by nuclear magnetic resonance spectroscopy in systemic lupus erythematosus.
- Regulation of tumor necrosis factor receptor-1 and the IKK-NF-kappaB pathway by LDL receptor-related protein explains the antiinflammatory activity of this receptor.
- Targeting cardiovascular risk in patients with diabetes: management of dyslipidemia.
Schools
University Of Tennessee Health Science Center College Of Medicine
Vanderbilt University
Doctors Specialties
- Endocrinology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Amerigroup (Wellpoint)
- Blue Card PPO
- Network S
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- LocalPlus
- Open Access Plus
- Coventry Health Care of Tennessee PPO
- Choice Care PPO
- Medicaid
- Medicare MCR
- PHCS Network PPO
- Tricare
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
Awards
- America's Top Doctors
- Nashville Lifestyles Magazine
- Elected Member
Education
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Vanderbilt University -
University of Tennessee College of Medicine
Hospital
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Vanderbilt Hospital and Clinics -
Williamson Medical Center
Drug Facts
| NPI NUMBER | 1194817502 | |
| NPPES Provider LastName | LINTON | |
| NPPES Provider FirstName | MACRAE | |
| NPPES Provider ZIPCode | 372320001 | |
| NPPES Provider State | TN | |
| Specialty Description | Endocrinology | |
| Total Claim Count | 316.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1194817502 |
| Last Name Of The Provider | LINTON |
| First Name Of The Provider | MACRAE |
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