Associate Professor in Pediatrics, Northwestern University Medical School

Allergy & Immunology

Ramsay Fuleihan works in the field of Allergy & Immunology. Ramsay L. Fuleihan is also a published doctor. He has 53 publication published. The most recent publication is: Calcium-dependent activation of TNF family gene expression by Ca2+/calmodulin kinase type IV/Gr and calcineurin.

Publications

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• Naturally occurring tolerance acquisition to foods in previously allergic children is characterized by antigen specificity and associated with increased subsets of reg...
• Cytokine responses to egg protein in previously allergic children who developed tolerance naturally.
• USIDNET: A Strategy to Build a Community of Clinical Immunologists.
• The Hyper IgM Syndromes.
• Primary immunodeficiency diseases: past, present and future.
• Chapter 27: Approach to primary immunodeficiency.
• The hygiene hypothesis revisited: does exposure to infectious agents protect us from allergy?
• DOCK8 deficiency, T cell receptor excision circles and newborn screening.
• Impaired B cell development and function in mice with a targeted disruption of the homeobox gene Hex.
• The hygiene hypothesis and atopic disease.
• CD40 ligand-deficient T cells from X-linked hyper-IgM syndrome carriers have intrinsic priming capability.
• Primary immunodeficiency: a never ending story.
• An RNA external guide sequence ribozyme targeting human interleukin-4 receptor alpha mRNA.
• Hyper-immunoglobulin M syndrome caused by a mutation in the promotor for CD40L.
• A 17-year-old boy with fever, sore throat, and headache.
• Gene silencing in the therapy of influenza and other respiratory diseases: Targeting to RNase P by use of External Guide Sequences (EGS).
• Use of intravenous immunoglobulin and adjunctive therapies in the treatment of primary immunodeficiencies: A working group report of and study by the Primary Immunodef...
• A dyslexia-associated variant in DCDC2 changes gene expression.
• Food allergy is associated with an increased risk of asthma.
• The hyper IgM syndrome.
• Cellular and molecular mechanisms of immune activation by microbial superantigens: studies using toxic shock syndrome toxin-1.
• Role of protein kinase activation in the induction of B cell adhesion by MHC class II ligands.
• Toxic shock syndrome toxin-1, toxic shock, and the immune system.
• Engagement of MHC-class II molecules by staphylococcal exotoxins delivers a comitogenic signal to human B cells.
• The epidemiology of tuberculosis patients admitted to Bhannes Hospital during the period extending from January 1980, till June 1983.
• Somatic mutation of human immunoglobulin V genes: bias, rate, and regulation.
• Role of CD40-CD40-ligand interaction in Ig-isotype switching.
• Localization of the genetic defect in X-linked immunoglobulin deficiency with normal or elevated IgM (HIGMX-1) to the CD40 ligand gene.
• Molecular pathology of X-linked immunoglobulin deficiency with normal or elevated IgM (HIGMX-1).
• Somatic mutation of human immunoglobulin V genes in the X-linked HyperIgM syndrome.
• CD40 ligand/CD40 deficiency.
• Expression of the CD40 ligand in T lymphocytes and induction of IgE isotype switching.
• A point mutation in exon 2 of the CD40 ligand gene causes the simultaneous expression of two defective mRNA species in X-linked hyperimmunoglobulinemia M.
• CD40-CD40 ligand (CD40L) interactions and X-linked hyperIgM syndrome (HIGMX-1).
• CD40 ligand expression is developmentally regulated in human thymocytes.
• Defective expression of the CD40 ligand in X chromosome-linked immunoglobulin deficiency with normal or elevated IgM.
• Cyclosporin A inhibits CD40 ligand expression in T lymphocytes.
• Decreased expression of the ligand for CD40 in newborn lymphocytes.
• Superantigens activate HIV-1 gene expression in monocytic cells.
• Deletions in the ligand for CD40 in X-linked immunoglobulin deficiency with normal or elevated IgM (HIGMX-1).
• Cross-linking of Fc gamma receptors activates HIV-1 long terminal repeat-driven transcription in human monocytes.
• X-linked agammaglobulinemia and immunoglobulin deficiency with normal or elevated IgM: immunodeficiencies of B cell development and differentiation.
• The X-linked hyperimmunoglobulin M syndrome.
• Allergy, immunology, and related disorders.
• Calcium-dependent activation of TNF family gene expression by Ca2+/calmodulin kinase type IV/Gr and calcineurin.
• Transfer of activation-dependent gene expression into T cell lines by recombinant adeno-associated virus.
• Human endothelial cells augment early CD40 ligand expression in activated CD4+ T cells through LFA-3-mediated stabilization of mRNA.
• When you hear hoof beats...do not forget the zebras.
• Transcriptional activity of the distal CD40 ligand promoter.
• A rare polyadenylation signal mutation of the FOXP3 gene (AAUAAA-->AAUGAA) leads to the IPEX syndrome.
• Hyper IgM syndrome: the other side of the coin.
• Genome-wide association study identifies peanut allergy-specific loci and evidence of epigenetic mediation in US children.

Schools

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• American University of Beirut Faculty of Medicine

• Duke University Med Center

• Duke University School Med

• Children39s Hospital Boston

Doctors Specialties

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• Pediatrics
• Allergy
• Immunology

Accepted Insurances

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Education

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• Boston Children's Hospital
• Duke University Hospital
• American University of Beirut Faculty of Medicine

Hospital

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• Lurie Children's Hospital of Chicago
• Northwestern Memorial Hospital
• Ann & Robert H. Lurie Children?

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