Professor, Neurology, UCLA School of Medicine
Neurology
Dr. Rhonda R. Voskuhl, MD
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Rhonda R. Voskuhl, MD practices Neurology. She went to Vanderbilt University School of Medicine. She received 3 awards: "Endowed Chair for MS Research", "Harry Weaver Award" and "Super Doctor". Rhonda Voskuhl is also a published MD. She has 82 paper published. The most recent paper is: Myelin protein expression in lymphoid tissues: implications for peripheral tolerance.
Publications
- Astrocyte CCL2 sustains immune cell infiltration in chronic experimental autoimmune encephalomyelitis.
- XY sex chromosome complement, compared with XX, in the CNS confers greater neurodegeneration during experimental autoimmune encephalomyelitis.
- Estrogen receptor-β ligand treatment after disease onset is neuroprotective in the multiple sclerosis model.
- Estrogen mediates neuroprotection and anti-inflammatory effects during EAE through ERα signaling on astrocytes but not through ERβ signaling on astrocytes or neurons.
- Therapeutic Testosterone Administration Preserves Excitatory Synaptic Transmission in the Hippocampus during Autoimmune Demyelinating Disease.
- The XX sex chromosome complement in mice is associated with increased spontaneous lupus compared with XY.
- Estrogen treatment prevents gray matter atrophy in experimental autoimmune encephalomyelitis.
- Dynamic Development of Glucocorticoid Resistance during Autoimmune Neuroinflammation.
- Estriol preserves synaptic transmission in the hippocampus during autoimmune demyelinating disease.
- Sex-related factors in multiple sclerosis susceptibility and progression.
- Neuroprotective effects of estrogens and androgens in CNS inflammation and neurodegeneration.
- Cortical atrophy in experimental autoimmune encephalomyelitis: In vivo imaging.
- Neuroprotection mediated through estrogen receptor-{alpha} in astrocytes.
- Hippocampal CA1 atrophy and synaptic loss during experimental autoimmune encephalomyelitis, EAE.
- Purkinje cell loss in experimental autoimmune encephalomyelitis.
- Estrogen treatment decreases matrix metalloproteinase (MMP)-9 in autoimmune demyelinating disease through estrogen receptor alpha (ERalpha).
- Reactive astrocytes form scar-like perivascular barriers to leukocytes during adaptive immune inflammation of the CNS.
- A role for sex chromosome complement in the female bias in autoimmune disease.
- Differential neuroprotective and antiinflammatory effects of estrogen receptor (ER)alpha and ERbeta ligand treatment.
- Testosterone treatment in multiple sclerosis: a pilot study.
- A female preponderance for chemically induced lupus in SJL/J mice.
- Cerebellar cortical atrophy in experimental autoimmune encephalomyelitis.
- Treatment with an estrogen receptor alpha ligand is neuroprotective in experimental autoimmune encephalomyelitis.
- A yin-yang effect between sex chromosome complement and sex hormones on the immune response.
- Method to detect functional estrogen receptor expression using estrogen receptor probing compound.
- Estrogen receptor alpha mediates estrogen's immune protection in autoimmune disease.
- Immune modulation in multiple sclerosis patients treated with the pregnancy hormone estriol.
- Comparison of multiple sclerosis lesions at 1.5 and 3.0 Tesla.
- Treatment of multiple sclerosis with the pregnancy hormone estriol.
- Gender issues and multiple sclerosis.
- Neuroprotective effects of testosterone treatment in men with multiple sclerosis.
- Testosterone replacement therapy for the treatment of neurological and neuropsychiatric disorders.
- Immune modulation and increased neurotrophic factor production in multiple sclerosis patients treated with testosterone.
- Estriol treatment ameliorates disease in males with experimental autoimmune encephalomyelitis: implications for multiple sclerosis.
- Dysregulation of IL-10 and IL-12p40 in secondary progressive multiple sclerosis.
- Androgens are protective in experimental autoimmune encephalomyelitis: implications for multiple sclerosis.
- Experimental autoimmune encephalomyelitis relapses are reduced in heterozygous golli MBP knockout mice.
- Estrogen and testosterone therapies in multiple sclerosis.
- Additive effects of combination treatment with anti-inflammatory and neuroprotective agents in experimental autoimmune encephalomyelitis.
- Estrogen treatment in multiple sclerosis.
- Neuroprotective and anti-inflammatory effects of estrogen receptor ligand treatment in mice.
- An animal model of cortical and callosal pathology in multiple sclerosis.
- Oestrogen receptor beta ligand: a novel treatment to enhance endogenous functional remyelination.
- Sex differences in autoimmune diseases.
- Estrogen receptor-β ligand treatment modulates dendritic cells in the target organ during autoimmune demyelinating disease.
- Do estroprogestinic hormones protect against chemotherapy-induced amenorrhea in multiple sclerosis?
- Onset of multiple sclerosis associated with anti-TNF therapy.
- Sex hormones in experimental autoimmune encephalomyelitis: implications for multiple sclerosis.
- Testosterone acts directly on CD4+ T lymphocytes to increase IL-10 production.
- "Classic" myelin basic proteins are expressed in lymphoid tissue macrophages.
- Mice resistant to experimental autoimmune encephalomyelitis have increased thymic expression of myelin basic protein and increased MBP specific T cell tolerance.
- Temporal kinetics and cellular phenotype of TNF p55/p75 receptors in experimental allergic encephalomyelitis.
- Decreased IL-12 production underlies the decreased ability of male lymph node cells to induce experimental autoimmune encephalomyelitis.
- Estriol ameliorates autoimmune demyelinating disease: implications for multiple sclerosis.
- Myelin protein expression in lymphoid tissues: implications for peripheral tolerance.
- Antisense knockdown of inducible nitric oxide synthase inhibits induction of experimental autoimmune encephalomyelitis in SJL/J mice.
- Myelin protein expression is increased in lymph nodes of mice with relapsing experimental autoimmune encephalomyelitis.
- Gender differences of inducible nitric oxide production in SJL/J mice with experimental autoimmune encephalomyelitis.
- Testosterone therapy ameliorates experimental autoimmune encephalomyelitis and induces a T helper 2 bias in the autoantigen-specific T lymphocyte response.
- Multiple sclerosis: re-expression of a developmental gene in chronic lesions correlates with remyelination.
- Exon 2 containing myelin basic protein (MBP) transcripts are expressed in lesions of experimental allergic encephalomyelitis (EAE).
- Antisense blockade of inducible nitric oxide synthase in glial cells derived from adult SJL mice.
- Epitope spreading occurs in active but not passive EAE induced by myelin basic protein.
- Human T lymphocytes specific for the immunodominant 83-99 epitope of myelin basic protein: recognition of golli MBP HOG 7.
- Strain-related differences in the ability of T lymphocytes to recognize proteins encoded by the golli-myelin basic protein gene.
- DR2/DQw1 inheritance and haplotype sharing in affected siblings from multiple sclerosis families.
- Gender differences in autoimmune demyelination in the mouse: implications for multiple sclerosis.
- Purification of immunologically active recombinant 21.5 kDa isoform of human myelin basic protein.
- T helper 1 (Th1) functional phenotype of human myelin basic protein-specific T lymphocytes.
- Myelin basic protein-specific T-cell responses in identical twins discordant or concordant for multiple sclerosis.
- A novel candidate autoantigen in a multiplex family with multiple sclerosis: prevalence of T-lymphocytes specific for an MBP epitope unique to myelination.
- A functional basis for the association of HLA class II genes and susceptibility to multiple sclerosis: cellular immune responses to myelin basic protein in a multiplex...
- T-lymphocyte recognition of a portion of myelin basic protein encoded by an exon expressed during myelination.
- T-cell receptor alpha and beta CDR3 expression by myelin basic protein-specific human T-cell clones.
- HLA restriction and TCR usage of T lymphocytes specific for a novel candidate autoantigen, X2 MBP, in multiple sclerosis.
- Experimental allergic encephalomyelitis induced by the peptide encoded by exon 2 of the MBP gene, a peptide implicated in remyelination.
- Diversity of T-cell receptor V alpha, V beta, and CDR3 expression by myelin basic protein-specific human T-cell clones.
- Sensory impairment in the hands secondary to spondylotic compression of the cervical spinal cord.
- HTLV-I-specific cytotoxic T lymphocytes in the cerebrospinal fluid of patients with HTLV-I-associated neurological disease.
- Hormone-based therapies in MS.
- Glial reactions and degeneration of myelinated processes in spinal cord gray matter in chronic experimental autoimmune encephalomyelitis.
Schools
Vanderbilt University
Dallas Co Hp Parkland Mem
Presbyterian Hospital
Doctors Specialties
- Neurology
Accepted Insurances
Awards
- Endowed Chair for MS Research
- Harry Weaver Award
- Super Doctor
Education
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National Institutes of Health Clinical Center -
Vanderbilt University School of Medicine
Hospital
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Ronald Reagan UCLA Medical Center
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