Physician
Pulmonology
Dr. Steven N. Mink, MD
Steven Mink is an expert in the field of Pulmonology. Steven N. Mink is a published physician. He published 71 publications, including: 'Histamine H3 activation depresses cardiac function in experimental sepsis.'
Publications
- Mechanisms of systemic vasodilation by lysozyme-c in septic shock.
- Benefits of ethyl gallate versus norepinephrine in the treatment of cardiovascular collapse in Pseudomonas aeruginosa septic shock in dogs.
- Ethyl gallate, a scavenger of hydrogen peroxide that inhibits lysozyme-induced hydrogen peroxide signaling in vitro, reverses hypotension in canine septic shock.
- Lysozyme, a mediator of sepsis that intrinsically generates hydrogen peroxide to cause cardiovascular dysfunction.
- N,N'-diacetylchitobiose, an inhibitor of lysozyme, reverses myocardial depression and lessens norepinephrine requirements in Escherichia coli sepsis in dogs.
- Lysozyme, a mediator of sepsis that produces vasodilation by hydrogen peroxide signaling in an arterial preparation.
- Lysozyme, a mediator of sepsis, impairs the cardiac neural adrenergic response by nonendothelial release of NO and inhibitory G protein signaling.
- Volume reduction surgery impairs immediate postoperative pulmonary function in canine emphysema.
- Characterization of membrane N-glycan binding sites of lysozyme for cardiac depression in sepsis.
- Lung volume reduction surgery in canine model of predominantly upper lobe emphysema: advantages of new surgical system.
- N,N',N"-triacetylglucosamine, an inhibitor of lysozyme, prevents myocardial depression in Escherichia coli sepsis in dogs.
- Pulmonary interstitial fibrosis as a presenting manifestation in perinuclear antineutrophilic cytoplasmic antibody microscopic polyangiitis.
- Blood pH level modulates organ metabolism of lactate in septic shock in dogs.
- Effect of histamine H3 receptor blockade on venous return and splanchnic hemodynamics in experimental bacteremia.
- Interdependence of flow between lobes reduces maximal emptying postresection in dogs.
- A comparison of fluticasone propionate 200 micrograms/day with beclomethasone dipropionate 400 micrograms/day in adult asthma.
- A mammary cell-specific enhancer in mouse mammary tumor virus DNA is composed of multiple regulatory elements including binding sites for CTF/NFI and a novel transcrip...
- Density-dependence of maximal expiratory flow and its correlation with small airway disease in smokers.
- Increased gravitational stress does not alter maximum expiratory flow.
- Hemodynamics in dogs with pulmonary hypertension due to emphysema.
- Mechanisms of increased maximum expiratory flow during HeO2 breathing in dogs.
- Mechanism of reduced maximum expiratory flow in methacholine-induced bronchoconstriction in dogs.
- Expiratory flow limitation and the response to breathing a helium-oxygen gas mixture in a canine model of pulmonary emphysema.
- Pulmonary function tests in the detection of small airway obstruction in a canine model of bronchiolitis obliterans.
- Plasma volume expansion in canine pneumococcal pneumonia: its effects on respiratory gas exchange and pneumonia size.
- Increased left ventricular stiffness impairs filling in dogs with pulmonary emphysema in respiratory failure.
- Interaction between effects of hypoxia and hypercapnia on altering left ventricular relaxation and chamber stiffness in dogs.
- Forced expiration and HeO2 response in canine peripheral airway obstruction.
- The effect of unilateral PEEP on gas exchange and pulmonary perfusion in canine lobar pneumonia.
- The physiology of recovery in experimental pneumococcal pneumonia.
- Dynamic lung function in dogs with compensatory lung growth.
- Aminophylline has little positive inotropic effect and a slightly negative diastolic effect on the left ventricle during hypoxic conditions in dogs.
- Left ventricular systolic performance is depressed in chronic pulmonary emphysema in dogs.
- Altered left ventricular chamber stiffness and isovolumic relaxation in dogs with chronic pulmonary hypertension caused by emphysema.
- Mechanism of reduced maximum expiratory flow in dogs with compensatory lung growth.
- Density dependence of maximal flow is lung volume dependent during bronchoconstriction.
- Doubling the dose of budesonide versus maintenance treatment in asthma exacerbations.
- Vascular and interstitial mechanics in canine pulmonary emphysema.
- Expiratory flow limitation in dogs with regional changes in lung mechanical properties.
- Effect of PEEP on gas exchange and pulmonary perfusion in canine lobar pneumonia.
- Pathophysiology of gas exchange and pulmonary perfusion in pneumococcal lobar pneumonia in dogs.
- Lung volume reduction surgery for the treatment of severe emphysema: a study in a single Canadian institution.
- Activation of pulmonary rapidly adapting receptors does not induce bronchoconstriction in dogs.
- Effect of pneumococcal lobar pneumonia on canine lung mechanics.
- Role of sodium in antigen-induced contraction of tracheal smooth muscle in dogs.
- Tumour necrosis factor-alpha, but not septic plasma depresses cardiac myofilament contraction.
- Does pentobarbital anesthesia depress left ventricular contractility in dogs?
- Insensitivity of maximum expiratory flow to bronchodilation in normal dogs.
- Impaired hepatic extraction and increased splanchnic production contribute to lactic acidosis in canine sepsis.
- Mechanism of lobar alveolar pressure decline during forced deflation in canine regional emphysema.
- Factors determining lobar emptying during maximal and partial forced deflations in nonhomogeneous airway obstruction in dogs.
- Heterogeneity of maximal lobar emptying rates in dogs with compensatory lung growth.
- How is maximal expiratory flow reduced in canine postpneumonectomy lung growth?
- Left ventricular contractility is depressed in IgE-mediated anaphylactic shock in dogs.
- Effect of continuous arteriovenous hemofiltration combined with systemic vasopressor therapy on depressed left ventricular contractility and tissue oxygen delivery in ...
- Effects of E. coli sepsis and myocardial depressant factor on interval-force relations in dog ventricle.
- hemofiltration reverses left ventricular dysfunction during sepsis in dogs.
- Constant infusion of epinephrine, but not bolus treatment, improves haemodynamic recovery in anaphylactic shock in dogs.
- Effects of anaphylaxis mediators on partitioned pulmonary vascular resistance during ragweed shock in dogs.
- Impaired sarcoplasmic calcium release inhibits myocardial contraction in experimental sepsis.
- Role of autacoids in cardiovascular colapse in anaphylactic shock in anaesthetized dogs.
- Histamine H3 receptor blockade improves cardiac function in canine anaphylaxis.
- Early but not delayed continuous arteriovenous hemofiltration improves cardiovascular function in sepsis in dogs.
- Effect of bolus epinephrine on systemic hemodynamics in canine anaphylactic shock.
- Histamine H3 activation depresses cardiac function in experimental sepsis.
- Atrial septal aneurysm with right-to-left interatrial shunting.
- Epinephrine fails to hasten hemodynamic recovery in fully developed canine anaphylactic shock.
- Sepsis causes presynaptic histamine H3 and alpha2-adrenergic dysfunction in canine myocardium.
- Lysozyme: a mediator of myocardial depression and adrenergic dysfunction in septic shock in dogs.
- Lysozyme binding to endocardial endothelium mediates myocardial depression by the nitric oxide guanosine 3'',5'' monophosphate pathway in sepsis.
Schools
Temple University
Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
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