Physician

Internal Medicine

Thomas Bersot works in the field of Internal Medicine. He graduated from Vanderbilt University School of Medicine. Thomas Bersot is a published doctor. He has 39 papers published. The most current publication "The -514 polymorphism in the hepatic lipase gene (LIPC) does not influence androgen-mediated stimulation of hepatic lipase activity." He accepts Medicare payments and is registered with Medicare.gov.

Publications

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• Genome-wide linkage and association analyses to identify genes influencing adiponectin levels: the GEMS Study.
• Comparison between Turkish Cardiovascular Risk Platform and United States National Cholesterol Education Program Adult Treatment Panel III definitions of the metabolic...
• Multiple QTLs influencing triglyceride and HDL and total cholesterol levels identified in families with atherogenic dyslipidemia.
• Modulation of high-density lipoproteins in a population in istanbul, Turkey, with low levels of high-density lipoproteins.
• Relation between atherogenic dyslipidemia and the Adult Treatment Program-III definition of metabolic syndrome (Genetic Epidemiology of Metabolic Syndrome Project).
• Risk determination of dyslipidemia in populations characterized by low levels of high-density lipoprotein cholesterol.
• Antibody phenotyping test for the human apolipoprotein E2 isoform.
• Sources of variability in genetic association studies: insights from the analysis of hepatic lipase (LIPC).
• Optimal waist:height ratio cut-off point for cardiometabolic risk factors in Turkish adults.
• Managing dyslipidemia in Turkey: suggested guidelines for a population characterized by low levels of high density lipoprotein cholesterol.
• High density lipoprotein cholesterol in coronary artery patients: is it as low as expected?
• Hypertriglyceridemia: management of atherogenic dyslipidemia.
• Serum insulin and inflammatory markers in overweight individuals with and without dyslipidemia.
• Genetic and phenotypic architecture of metabolic syndrome-associated components in dyslipidemic and normolipidemic subjects: the GEMS Study.
• Analysis of agreement among definitions of metabolic syndrome in nondiabetic Turkish adults: a methodological study.
• Anthropometric indices and their relationship with cardiometabolic risk factors in a sample of Turkish adults.
• The -514 polymorphism in the hepatic lipase gene (LIPC) does not influence androgen-mediated stimulation of hepatic lipase activity.
• Elevated hepatic lipase activity and low levels of high density lipoprotein in a normotriglyceridemic, nonobese Turkish population.
• Low levels of high density lipoproteins in Turks, a population with elevated hepatic lipase. High density lipoprotein characterization and gender-specific effects of a...
• Plasma lipids in Turkish children: impact of puberty, socioeconomic status, and nutrition on plasma cholesterol and HDL.
• A unique haplotype of the apolipoprotein B-100 allele associated with familial defective apolipoprotein B-100 in a Chinese man discovered during a study of the prevale...
• Familial defective apolipoprotein B-100: a mutation of apolipoprotein B that causes hypercholesterolemia.
• Type III hyperlipoproteinemia associated with apolipoprotein E phenotype E3/3. Structure and genetics of an apolipoprotein E3 variant.
• Identification of a new structural variant of human apolipoprotein E, E2(Lys146 leads to Gln), in a type III hyperlipoproteinemic subject with the E3/2 phenotype.
• Type III hyperlipoproteinemia: a focus on lipoprotein receptor-apolipoprotein E2 interactions.
• Alterations in human high-density lipoproteins, with or without increased plasma-cholesterol, induced by diets high in cholesterol.
• A-Imilano apoprotein. Isolation and characterization of a cysteine-containing variant of the A-I apoprotein from human high density lipoproteins.
• Receptor binding activity of high-density lipoproteins containing apoprotein E from abetalipoproteinemic and normal neonate plasma.
• Interaction of swine lipoproteins with the low density lipoprotein receptor in human fibroblasts.
• Apoprotein (E--A-II) complex of human plasma lipoproteins. II. Receptor binding activity of a high density lipoprotein subfraction modulated by the apo(E--A-II) complex.
• Cholesteryl ester accumulation in mouse peritoneal macrophages induced by beta-migrating very low density lipoproteins from patients with atypical dysbetalipoproteinemia.
• Identity of very low density lipoprotein apoproteins of plasma and liver Golgi apparatus.
• Fat feeding in humans induces lipoproteins of density less than 1.006 that are enriched in apolipoprotein [a] and that cause lipid accumulation in macrophages.
• Purification and characterization of human lipoprotein lipase and hepatic triglyceride lipase. Reactivity with monoclonal antibodies to hepatic triglyceride lipase.
• Isolation and characterization of an apoprotein from the d less than 1.006 lipoproteins of human and canine lymph homologous with the rat A-IV apoprotein.
• Apolipoprotein A-IMilano. Detection of normal A-I in affected subjects and evidence for a cysteine for arginine substitution in the variant A-I.
• Characterization of the rat apolipoproteins. I. The low molecular weight proteins of rat plasma high density lipoproteins.
• Atypical familial dysbetalipoproteinemia associated with apolipoprotein phenotype E3/3.

Schools

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• Vanderbilt University

Doctors Specialties

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• Internal Medicine

Accepted Insurances

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Education

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• National Institutes of Health Clinical Center
• UCSF
• Vanderbilt University School of Medicine

Hospital

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• San Francisco VA Medical Center
• UCSF Medical Center

Drug Facts

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NPI NUMBER		1205037629
NPPES Provider LastName		BERSOT
NPPES Provider FirstName		THOMAS
NPPES Provider ZIPCode		941103518
NPPES Provider State		CA
Specialty Description		Endocrinology
Total Claim Count		195.0
Distinct Opioid Count		0.0
Opioid Claim Count		0.0
Percent Opioid Claims		0.0

Helpful Reviews

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Medicare Facts

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National Provider Identifier [NPI]	1205037629
Last Name Of The Provider	BERSOT
First Name Of The Provider	THOMAS
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