Associate Professor of , UCSF
Neurology
Dr. Raymond A. Swanson, MD
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Raymond Swanson practices Neurology. He graduated from University of Michigan Medical School. Raymond Swanson is a published MD as well. He has 99 publications published. The latest was: Can experimental conditions explain the discrepancy over glutamate stimulation of aerobic glycolysis?
Publications
- Neuronal Glutathione Content and Antioxidant Capacity can be Normalized In Situ by N-acetyl Cysteine Concentrations Attained in Human CerebroSpinal Fluid.
- Tat-NR2B9c prevents excitoToxic neuronal superoxide production.
- Cerebrospinal fluid concentrations of N-acetylcysteine after oral administration in Parkinson's Disease.
- Mitochondrial reactive oxygen species are scavenged by Cockayne Syndrome B protein in human fibroblasts without nuclear DNA damage.
- Opposing Effects of Glucose on Stroke and Reperfusion Injury: Acidosis, Oxidative Stress, and Energy Metabolism.
- NADPH Oxidase-2: Linking Glucose, Acidosis, and ExcitoToxicity in Stroke.
- Targeting Microglial Activation in Stroke Therapy: Pharmacological Tools and Gender Effects.
- Glucose, acid and aspartate: Friends and foes of the axon.
- Intracellular pH reduction prevents excitoToxic and ischemic neuronal death by inhibiting NADPH oxidase.
- Mitochondrial Dysfunction Induced by Nuclear Poly(ADP-Ribose) Polymerase-1: a Treatable Cause of Cell Death in Stroke.
- Poly(ADP-ribose) polymerase-1-induced NAD(+) depletion promotes nuclear factor-?B transcriptional activity by preventing p65 de-acetylation.
- Prevention of traumatic brain injury-induced neuron death by intranasal delivery of nicotinamide adenine dinucleotide.
- One cell's poison is another cell's cure.
- Poly(ADP-ribose)polymerase-1 modulates microglial responses to amyloid beta.
- Hyperglycemia promotes tissue plasminogen activator-induced hemorrhage by Increasing superoxide production.
- Nuclear factor erythroid 2-related factor 2 facilitates neuronal glutathione synthesis by upregulating neuronal excitatory amino Acid transporter 3 expression.
- N-acetylcysteine prevents loss of dopaminergic neurons in the EAAC1-/- mouse.
- N-acetylcysteine prevents loss of dopaminergic neurons in the EAAC1(-/-) mouse.
- EAAC1 gene deletion alters zinc homeostasis and exacerbates neuronal injury after transient cerebral ischemia.
- Microglial activation in Stroke: therapeutic targets.
- Astrocytic poly(ADP-ribose) polymerase-1 activation leads to bioenergetic depletion and inhibition of glutamate uptake capacity.
- NAD+ depletion is necessary and sufficient for poly(ADP-ribose) polymerase-1-mediated neuronal death.
- Pyruvate protects against experimental stroke via an anti-Inflammatory mechanism.
- NADPH oxidase is the primary source of superoxide induced by NMDA receptor activation.
- Astrocyte cultures exhibit P2X7 receptor channel opening in the absence of exogenous ligands.
- Inhibition of poly(ADP-ribose) polymerase suppresses inflammation and promotes recovery after ischemic injury.
- Selective down-regulation of nuclear poly(ADP-ribose) glycohydrolase.
- Glucose and NADPH oxidase drive neuronal superoxide formation in Stroke.
- Sequential release of nitric oxide, zinc, and superoxide in hypoglycemic neuronal death.
- Zinc triggers microglial activation.
- Differences among cell types in NAD(+) compartmentalization: a comparison of neurons, astrocytes, and cardiac myocytes.
- Hypoglycemia, brain energetics, and hypoglycemic neuronal death.
- Zinc inhibits astrocyte glutamate uptake by activation of poly(ADP-ribose) polymerase-1.
- Astrocyte glycogen sustains neuronal activity during hypoglycemia: studies with the glycogen phosphorylase inhibitor CP-316,819 ([R-R*,S*]-5-chloro-N-[2-hydroxy-3-(met...
- Hypoglycemic neuronal death is triggered by glucose reperfusion and activation of neuronal NADPH oxidase.
- Pyruvate improves recovery after PARP-1-associated energy failure induced by oxidative stress in neonatal rat cerebrocortical slices.
- Use of a poly(ADP-ribose) polymerase inhibitor to suppress inflammation and neuronal death after cerebral ischemia-reperfusion.
- INTRODUCTION: Immune mechanisms of neurodegeneration.
- Intracerebral hematoma: beyond the mass lesion.
- Poly(ADP-ribose) polymerase-1 hyperactivation and impairment of mitochondrial respiratory chain complex I function in reperfused mouse hearts.
- Minocycline inhibits poly(ADP-ribose) polymerase-1 at nanomolar concentrations.
- Direct phosphorylation and regulation of poly(ADP-ribose) polymerase-1 by extracellular signal-regulated kinases 1/2.
- Neuronal glutathione deficiency and age-dependent neurodegeneration in the EAAC1 deficient mouse.
- Poly(ADP-ribose) polymerase-1 activation in a primate model of Multiple sclerosis.
- Ischemia-induced programmed cell death in astrocytes.
- Pyruvate administered after severe hypoglycemia reduces neuronal death and cognitive impairment.
- Acidosis causes endoplasmic reticulum stress and caspase-12-mediated astrocyte death.
- Poly(ADP-ribose) polymerase-1 promotes microglial activation, proliferation, and matrix metalloproteinase-9-mediated neuron death.
- Hypoglycemia induces transient neurogenesis and subsequent progenitor cell loss in the rat hippocampus.
- NAD+ as a metabolic link between DNA damage and cell death.
- Zinc release contributes to hypoglycemia-induced neuronal death.
- Using 31P NMR spectroscopy at 14.1 Tesla to investigate PARP-1 associated energy failure and metabolic rescue in cerebrocortical slices.
- Vitamin B12 deficiency.
- Poly(ADP-ribose) polymerase-1-mediated cell death in astrocytes requires NAD+ depletion and mitochondrial permeability transition.
- Alternative, nonapoptotic programmed cell death: mediation by arrestin 2, ERK2, and Nur77.
- Human serum albumin and its N-terminal tetrapeptide (DAHK) block oxidant-induced neuronal death.
- Hypoglycemic neuronal death and cognitive impairment are prevented by poly(ADP-ribose) polymerase inhibitors administered after hypoglycemia.
- Overexpression of rat heat shock protein 70 reduces neuronal injury after transient focal ischemia, transient global ischemia, or kainic acid-induced seizures.
- NAD+ repletion prevents PARP-1-induced glycolytic blockade and cell death in cultured mouse astrocytes.
- Ischemic preconditioning by caspase cleavage of poly(ADP-ribose) polymerase-1.
- Cerebral ischemia and infarction from atheroemboli <100 microm in Size.
- Overexpression of rat heat shock protein 70 is associated with reduction of early mitochondrial cytochrome C release and subsequent DNA fragmentation after permanent f...
- P2X7 receptor-mediated release of excitatory amino acids from astrocytes.
- Astrocytes and brain injury.
- Barbiturates induce mitochondrial depolarization and potentiate excitoToxic neuronal death.
- Tricarboxylic acid cycle substrates prevent PARP-mediated death of neurons and astrocytes.
- Beneficial effects of minocycline and botulinum toxin-induced constraint physical therapy following experimental traumatic brain injury.
- Sirtuins in neurodegenerative Diseases: an update on potential mechanisms.
- Phosphoinositide 3-kinase couples NMDA receptors to superoxide release in excitoToxic neuronal death.
- A conserved ubiquitin ligase of the nuclear envelope/endoplasmic reticulum that functions in both ER-associated and Matalpha2 repressor degradation.
- A fosmid-based genomic map and identification of 474 genes of the hyperthermophilic archaeon Pyrobaculum aerophilum.
- Species variants of the IsK protein: differences in kinetics, voltage dependence, and La3+ block of the currents expressed in Xenopus oocytes.
- Resolution of Michaelis complex, acylation, and conformational change steps in the reactions of the serpin, plasminogen activator inhibitor-1, with tissue plasminogen ...
- Specific covalent labeling of cytochrome P-450CAM with 1-(4-azidophenyl)imidazole, an inhibitor-derived photoaffinity probe for P-450 heme proteins.
- A prospective randomized study of neurocognitive function and s-100 protein after antegrade or retrograde brain perfusion with hypothermic arrest for aortic surgery.
- Selective suppression of cytochrome P-450 gene expression by interleukins 1 and 6 in rat liver.
- Glucose and infarct size.
- Do CK-MB results affect chest Pain decision making in the emergency department?
- Cloning and expression of the delayed-rectifier IsK channel from neonatal rat heart and diethylstilbestrol-primed rat uterus.
- K+ currents expressed from the guinea pig cardiac IsK protein are enhanced by activators of protein kinase C.
- Post-traumatic epidermoid cyst presenting with Headache.
- The stress gene response in brain.
- Intravenous heparin for acute Stroke: what can we learn from the megatrials?
- Can experimental conditions explain the Discrepancy over glutamate stimulation of aerobic glycolysis?
- BW619C89, a glutamate release inhibitor, protects against focal cerebral ischemic damage.
- Infarct measurement methodology.
- Alcohol exacerbates behavioral and neurochemical effects of rat Spinal cord trauma.
- Amnestic Syndrome and vertical gaze palsy: early detection of bilateral thalamic infarction by CT and NMR.
- Metabolic mapping with cellular resolution: c-fos vs. 2-deoxyglucose.
- Female sex hormones inhibit volume regulation in rat brain astrocyte culture.
- The regional distribution of glycogen in rat brain fixed by microwave irradiation.
- Regional brain glycogen stores and metabolism during complete global ischaemia.
- Effects of L-glutamate, D-aspartate, and monensin on glycolytic and oxidative glucose metabolism in mouse astrocyte cultures: further evidence that glutamate uptake is...
- Sensory stimulation induces local cerebral glycogenolysis: demonstration by autoradiography.
- Astrocytic glycogen influences axon function and survival during glucose deprivation in central white matter.
- Astrocyte glucose metabolism under normal and pathological conditions in vitro.
- Phencyclidine induction of the hsp 70 stress gene in injured pyramidal neurons is mediated via Multiple receptors and voltage gated calcium channels.
- HIV-1 envelope gp120 alters astrocytes in human brain cultures.
Schools
University Of Michigan Medical School
Conditions Treated
- Alzheimer's Disease
- Ataxia
- Carpal Tunnel Syndrome
- Cerebrovascular Disease
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Doctors Specialties
- Neurology
Accepted Insurances
Education
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UCSF -
Virginia Mason Medical Center -
University of Michigan Medical School
Hospital
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San Francisco VA Medical Center -
UCSF Medical Center
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